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Please use this identifier to cite or link to this item: https://dspace.bsmu.edu.ua/handle/123456789/26464
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dc.contributor.authorKmet, Olga G.-
dc.contributor.authorFilipets, Nataliia D.-
dc.contributor.authorYaremii, Iryna M.-
dc.contributor.authorKmet, Taras I.-
dc.contributor.authorVepriuk, Yurii M.-
dc.contributor.authorHrachova, Tamara I.-
dc.date.accessioned2025-04-08T12:07:28Z-
dc.date.available2025-04-08T12:07:28Z-
dc.date.issued2020-
dc.identifier.otherDOI: 10.31688/ABMU.2020.55.1.01-
dc.identifier.urihttps://dspace.bsmu.edu.ua/handle/123456789/26464-
dc.description.abstractThe objective of the study was to evaluate the protective effect of carbacetam under conditions of mitochondrial dysfunction of the brain in rats with scopolamine-induced Alzheimer’s disease. Conclusions. Reduced intensity of mitochondrial swelling, improvement of the antioxidant system state and energy supply of mitochondria in the cerebral cortex and hippocampus of rats with scopolamine-induced Alzheimer’s disease are indicative of mitoprotective properties of carbacetam as a promising neuroprotector.uk_UA
dc.language.isoenuk_UA
dc.publisherArchives of the Balkan Medical Union (міжнародний рецензований міждисциплінарний журнал)uk_UA
dc.subjectcarbacetamuk_UA
dc.subjectscopolamine-induced Alzheimer’s diseaseuk_UA
dc.subjectfunctional state of the mitochondriauk_UA
dc.titleExperimental assessment of carbacetam effect on the cerebral mitochondria in rats with scopolamine-induced Alzheimer’s diseaseuk_UA
dc.typeArticleuk_UA
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